肥厚型心肌病的心电图特征(2)
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The pathophysiological mechanisms of the Q wave in HCM are not fully understood; however, it is assumed that it may be related to the ratio of the upper anterior ptal thickness and other regions of the LVs and right ventricles (RVs). Wider Q waves in this subt of patients are reported to be associated with late gadolinium enhancement (LGE).In 5% of the patients, the ECG may be normal especially in the early phas of the dia.
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代表希望的花Figure 6.4 illustrates typical ECG (panel A), long-axis Echo (panel B), and magnetic resonance imaging(MRI) of a patient with HCM (panel C).
翻 译
桑螵蛸的功效与作用员工大会发言稿肥厚型心肌病Q波的病理生理机制尚未完全明确;目前推测其可能与室间隔前上厚度与左室其他区域和右心室室壁厚度的比值有关。在这组患者中,宽Q波与钆造影剂延迟强化有关。5%患者心电图可能是正常的,尤其在疾病的早期阶段。
图6.4显示了肥厚型心肌病患者的典型心电图(A),超声心脏长轴(B)和核磁共振(C)。
图6-4 HCM患者的心电图、超声和磁共振成像。A:HCM患者的12导联心电图显示V3-V6导联和I、II、III和aVF导联有明显的负向T波。B:同一患者的超声心动图,胸骨旁长轴切面显示明显的室间隔肥厚,室间隔厚度19mm,后壁厚度4mm。C.心脏磁共振成像长轴切面显示室间隔肥厚。
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英文原文:
心灵家园聊天室The most important differential diagnosis is athlete’s heart and LVH due to hypertension (e Chapter 4). In this ca, other noninvasive imaging techniques, such as Echo and cardiac MRI, are valuable. HCM patients show an unusual pattern of LV hypertrophy with a small LV cavity (<35mm),>abnormal LV filling, positive family history, abnormal genetic profile, whereas patients with athletic heart demonstrate LV cavity of >55mmHg, have normal LV filling, no family history, and negative HCM genetic profile. Bizarre ECG patterns and LAE are often en in athletic individuals.Interestingly, with detraining,most of the changes will normalize in athletes.
翻 译
最重要的鉴别诊断是运动员的心脏和高血压导致的左心室肥厚(前文有讲述)。这种情况下,其他非侵入性成像技术,如超声和心脏核磁,是有价值的。肥厚型心肌病患者左室肥厚,心室腔小(<35mm),左房扩大,左室充盈异常,有家族史和基因异常,而运动员的心脏左心室腔>55mm,左室充盈正常,无家族病史,肥厚型心肌病基因阴性。运动员心电图常有左房扩大和其他一些少见改变。有趣的是,随着训练的减少,大部分的变化将会恢复正常。(翻译:王世聪;校对:何树楠)
不做就图论图,只做透彻解读!
病因甄别、合并疾病、
孕妇鸽子汤
体格检查、可能诊断、治疗策略……
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